Thank you Dr Fathi for the great explanation. The final Dx was mid-LAD occlusion.

this is the explanation from the source:-

Take-Home Points 

• T wave symmetry: most important parameter for distinguishing between abnormal and normal T waves (not T wave amplitude)Normal/non-pathologic T waves → asymmetric(upstroke ≠ downstroke)
• Abnormal/pathologic (“hyperacute”) → symmetric(upstroke = downstroke)
• Causes of symmetric (abnormal) T waves (whether tall or not): hyperkalemia, early phase of acute myocardial infarction (MI)
• EKG findings in early phase of acute MI:
• 1) Hyperacute T waves
• Cause: Ischemic damage to myocardium leads to malfunction of the Na+/K+ ATPase → Na+ remains in the cell, K+remains outside the cell → causes local extracellular hyperkalemia, causing peaked T waves
• 2) Short ST segment
• Cause: increased intracellular Na+ leads to reversal of the Na+/Ca2+ exchanger →pumps out Na+, pumps in Ca2+ → causes intracellular hypercalcemia, causing short ST segment
• 3) deWinter’s sign: upsloping ST depressions plus hyperacute T waves in the precordial leadsSpecific for early phase of acute MI due to LAD occlusion